Choline Magnesium Trisalicylate and Weed
Edited by Hugh Soames
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Choline Magnesium Trisalicylate and Weed
Most people who consume marijuana do so for its mood-altering and relaxing abilities. Weed gives people a high and allows them to relax. However, heavy consumption of weed can cause unwanted results. It can increase the anxiety and depression a person experiences, and it can interact with certain other drugs including Choline Magnesium Trisalicylate. It is important to remember that interactions do occur with all types of drugs, to a great or lesser extent and this article details the interactions of mixing Choline Magnesium Trisalicylate and Weed.
Mixing Choline Magnesium Trisalicylate and Weed
Salicylic acid is an organic compound with the formula HOC6H4COOH. A colorless, bitter-tasting solid, it is a precursor to and a metabolite of aspirin (acetylsalicylic acid). It is a plant hormone, and has been listed by the EPA Toxic Substances Control Act (TSCA) Chemical Substance Inventory as an experimental teratogen. The name is from Latin salix for willow tree, from which it was initially identified and derived. It is an ingredient in some anti-acne products. Salts and esters of salicylic acid are known as salicylates.
Salicylic acid as a medication is commonly used to remove the outer layer of the skin. As such, it is used to treat warts, psoriasis, acne vulgaris, ringworm, dandruff, and ichthyosis.
Similar to other hydroxy acids, salicylic acid is an ingredient in many skincare products for the treatment of seborrhoeic dermatitis, acne, psoriasis, calluses, corns, keratosis pilaris, acanthosis nigricans, ichthyosis, and warts.
Salicylic acid is used as a food preservative, a bactericide, and an antiseptic.
Salicylic acid is used in the production of other pharmaceuticals, including 4-aminosalicylic acid, sandulpiride, and landetimide (via salethamide).
Salicylic acid has long been a key starting material for making acetylsalicylic acid (aspirin). Aspirin (acetylsalicylic acid or ASA) is prepared by the esterification of the phenolic hydroxyl group of salicylic acid with the acetyl group from acetic anhydride or acetyl chloride. ASA is the standard to which all the other non-steroidal anti-inflammatory drugs (NSAIDs) are compared. In veterinary medicine, this group of drugs is mainly used for treatment of inflammatory musculoskeletal disorders.
Bismuth subsalicylate, a salt of bismuth and salicylic acid, “displays anti-inflammatory action (due to salicylic acid) and also acts as an antacid and mild antibiotic”. It is the active ingredient in stomach-relief aids such as Pepto-Bismol and some formulations of Kaopectate.
Other derivatives include methyl salicylate used as a liniment to soothe joint and muscle pain and choline salicylate used topically to relieve the pain of mouth ulcers. Aminosalicylic acid is used to induce remission in ulcerative colitis, and has been used as an antitubercular agent often administered in association with isoniazid.
Sodium salicylate is a useful phosphor in the vacuum ultraviolet spectral range, with nearly flat quantum efficiency for wavelengths between 10 and 100 nm. It fluoresces in the blue at 420 nm. It is easily prepared on a clean surface by spraying a saturated solution of the salt in methanol followed by evaporation.[citation needed]
Salicylic acid modulates COX-1 enzymatic activity to decrease the formation of pro-inflammatory prostaglandins. Salicylate may competitively inhibit prostaglandin formation. Salicylate’s antirheumatic (nonsteroidal anti-inflammatory) actions are a result of its analgesic and anti-inflammatory mechanisms.
Salicylic acid, when applied to the skin surface, works by causing the cells of the epidermis to slough off more readily, preventing pores from clogging up, and allowing room for new cell growth. Salicylic acid inhibits the oxidation of uridine-5-diphosphoglucose (UDPG) competitively with nicotinamide adenine dinucleotide and noncompetitively with UDPG. It also competitively inhibits the transferring of glucuronyl group of uridine-5-phosphoglucuronic acid to the phenolic acceptor.
The wound-healing retardation action of salicylates is probably due mainly to its inhibitory action on mucopolysaccharide synthesis.
If high concentrations of salicylic ointment are used topically, high levels of salicylic acid can enter the blood, requiring hemodialysis to avoid further complications.
Despite the potential teratogenic risk of salicylic acid, cosmetic applications of the drug pose no significant risk. Even in a worst-case use scenario in which one was using multiple salicylic acid containing topical products, the aggregate plasma concentration of salicylicic acid was well below what was permissible for acetylsalicylic acid (aspirin). Since oral aspirin (which produces much higher salicylic acid plasma concentrations than dermal salicylic acid applications) poses no significant adverse pregnancy outcomes in terms of frequency of stillbirth, birth defects or developmental delay, use of salicylic acid containing cosmetics is safe for pregnant women.
Salicylic acid is biosynthesized from the amino acid phenylalanine. In Arabidopsis thaliana, it can be synthesized via a phenylalanine-independent pathway.
Commercial vendors prepare sodium salicylate by treating sodium phenolate (the sodium salt of phenol) with carbon dioxide at high pressure (100 atm) and high temperature (115 °C) – a method known as the Kolbe-Schmitt reaction. Acidifying the product with sulfuric acid gives salicylic acid:
At the laboratory scale, it can also be prepared by the hydrolysis of aspirin (acetylsalicylic acid) or methyl salicylate (oil of wintergreen) with a strong acid or base; these reactions reverse those chemicals’ commercial syntheses.
Upon heating, salicylic acid converts to phenyl salicylate:
Further heating gives xanthone.
Salicylic acid as its conjugate base is a chelating agent, with an affinity for iron(III).
Salicylic acid slowly degrades to phenol and carbon dioxide at 200–230 °C:
Willow has long been used for medicinal purposes. Dioscorides, whose writings were highly influential for more than 1,500 years, used ‘Itea’ (which was possibly a species of willow) as a treatment for ‘painful intestinal obstructions,’ birth control, for ‘those who spit blood,’ to remove calluses and corns and, externally, as a ‘warm pack for gout.’ William Turner, in 1597, repeated this, saying that willow bark, ‘being burnt to ashes, and steeped in vinegar, takes away corns and other like risings in the feet and toes.’ Some of these cures may describe the action of salicylic acid, which can be derived from the salicin present in willow. It is, however, a modern myth that Hippocrates used willow as a painkiller.
Hippocrates, Galen, Pliny the Elder, and others knew that decoctions containing salicylate could ease pain and reduce fevers.
It was used in Europe and China to treat these conditions. This remedy is mentioned in texts from Ancient Egypt, Sumer, and Assyria.
The Cherokee and other Native Americans use an infusion of the bark for fever and other medicinal purposes. In 2014, archaeologists identified traces of salicylic acid on seventh-century pottery fragments found in east-central Colorado.
The Reverend Edward Stone, a vicar from Chipping Norton, Oxfordshire, England, reported in 1763 that the bark of the willow was effective in reducing a fever.
An extract of willow bark, called salicin, after the Latin name for the white willow (Salix alba), was isolated and named by German chemist Johann Andreas Buchner in 1828. A larger amount of the substance was isolated in 1829 by Henri Leroux, a French pharmacist. Raffaele Piria, an Italian chemist, was able to convert the substance into a sugar and a second component, which on oxidation becomes salicylic acid.
Salicylic acid was also isolated from the herb meadowsweet (Filipendula ulmaria, formerly classified as Spiraea ulmaria) by German researchers in 1839. Their extract caused digestive problems such as gastric irritation, bleeding, diarrhea, and even death when consumed in high doses.
In 1874 the Scottish physician Thomas MacLagan experimented with salicin as a treatment for acute rheumatism, with considerable success, as he reported in The Lancet in 1876. Meanwhile, German scientists tried sodium salicylate with less success and more severe side effects.
In 1979, salicylates were found to be involved in induced defenses of tobacco against tobacco mosaic virus. In 1987, salicylic acid was identified as the long-sought signal that causes thermogenic plants, such as the voodoo lily, Sauromatum guttatum, to produce heat.
Salicylic acid occurs in plants as free salicylic acid and its carboxylated esters and phenolic glycosides. Several studies suggest that humans metabolize salicylic acid in measurable quantities from these plants. High-salicylate beverages and foods include beer, coffee, tea, numerous fruits and vegetables, sweet potato, nuts, and olive oil. Meat, poultry, fish, eggs, dairy products, sugar, breads and cereals have low salicylate content.
Some people with sensitivity to dietary salicylates may have symptoms of allergic reaction, such as bronchial asthma, rhinitis, gastrointestinal disorders, or diarrhea, so may need to adopt a low-salicylate diet.
Salicylic acid is a phenolic phytohormone, and is found in plants with roles in plant growth and development, photosynthesis, transpiration, and ion uptake and transport. Salicylic acid is involved in endogenous signaling, mediating plant defense against pathogens. It plays a role in the resistance to pathogens (i.e. systemic acquired resistance) by inducing the production of pathogenesis-related proteins and other defensive metabolites. SA’s defense signaling role is most clearly demonstrated by experiments which do away with it: Delaney et al. 1994, Gaffney et al. 1993, Lawton et al. 1995, and Vernooij et al. 1994 each use Nicotiana tabacum or Arabidopsis expressing nahG, for salicylate hydroxylase. Pathogen inoculation did not produce the customarily high SA levels, SAR was not produced, and no PR genes were expressed in systemic leaves. Indeed, the subjects were more susceptible to virulent – and even normally avirulent – pathogens.
Exogenously, salicylic acid can aid plant development via enhanced seed germination, bud flowering, and fruit ripening, though too high of a concentration of salicylic acid can negatively regulate these developmental processes.
The volatile methyl ester of salicylic acid, methyl salicylate, can also diffuse through the air, facilitating plant-plant communication. Methyl salicylate is taken up by the stomata of the nearby plant, where it can induce an immune response after being converted back to salicylic acid.
A number of proteins have been identified that interact with SA in plants, especially salicylic acid binding proteins (SABPs) and the NPR genes (nonexpressor of pathogenesis-related genes), which are putative receptors.
Research has found that anxiety is one of the leading symptoms created by marijuana in users, and that there is a correlation between Choline Magnesium Trisalicylate and Weed and an increase in anxiety.
Anyone mixing Choline Magnesium Trisalicylate and weed is likely to experience side effects. This happens with all medications whether weed or Choline Magnesium Trisalicylate is mixed with them. Side effects can be harmful when mixing Choline Magnesium Trisalicylate and weed. Doctors are likely to refuse a patient a Choline Magnesium Trisalicylate prescription if the individual is a weed smoker or user. Of course, this could be due to the lack of studies and research completed on the mixing of Choline Magnesium Trisalicylate and Weed.
Heavy, long-term weed use is harmful for people. It alters the brain’s functions and structure, and all pharmaceuticals and drugs including Choline Magnesium Trisalicylate are designed to have an impact on the brain. There is a misplaced belief that pharmaceuticals and medication work by treating only the parts of the body affected yet this is obviously not the case in terms of Choline Magnesium Trisalicylate. For example, simple painkiller medication does not heal the injury, it simply interrupts the brains functions to receive the pain cause by the injury. To say then that two drugs, Choline Magnesium Trisalicylate and Weed, dol not interact is wrong. There will always be an interaction between Choline Magnesium Trisalicylate and Weed in the brain11.J. D. Brown and A. G. Winterstein, Potential Adverse Drug Events and Drug–Drug Interactions with Medical and Consumer Cannabidiol (CBD) Use – PMC, PubMed Central (PMC).; Retrieved September 27, 2022, from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6678684/.
One of the milder side effects of mixing Choline Magnesium Trisalicylate and Weed is Scromiting. This condition, reportedly caused by mixing Choline Magnesium Trisalicylate and Weed, describes a marijuana-induced condition where the user experiences episodes of violent vomiting, which are often so severe and painful that they cause the person to scream. The medical term for Scromiting by mixing Choline Magnesium Trisalicylate and Weed is cannabinoid hyperemesis syndrome, or CHS. For these reasons, some people choose to quit smoking weed.
It was first included in scientific reports in 2004. Since then, researchers have determined that Scromiting is the result of ongoing, long-term use of marijuana—particularly when the drug contains high levels of THC, marijuana’s main psychoactive ingredient. Some experts believe that the receptors in the gut become overstimulated by THC, thus causing the repeated cycles of vomiting.
In the long run, a person can become even more depressed. There is a belief that marijuana is all-natural and not harmful to a person’s health. This is not true and Choline Magnesium Trisalicylate and weed can cause health issues the more a person consumes it.
How does Weed effect the potency of Choline Magnesium Trisalicylate?
The way in which the body absorbs and process Choline Magnesium Trisalicylate may be affected by weed. Therefore, the potency of the Choline Magnesium Trisalicylate may be less effective. Marijuana inhibits the metabolization of Choline Magnesium Trisalicylate. Not having the right potency of Choline Magnesium Trisalicylate means a person may either have a delay in the relief of their underlying symptoms.
A person seeking Choline Magnesium Trisalicylate medication that uses weed should speak to their doctor. It is important the doctor knows about a patient’s weed use, so they can prescribe the right Choline Magnesium Trisalicylate medication and strength. Or depending on level of interactions they may opt to prescribe a totally different medication. It is important for the doctor to know about their patient’s marijuana use. Weed is being legalized around the US, so doctors should be open to speaking about a patient’s use of it.
Sideffects of Choline Magnesium Trisalicylate and Weed
Many individuals may not realize that there are side effects and consequences to mixing Choline Magnesium Trisalicylate and Weed such as:
- Dizziness
- Sluggishness
- Drowsiness
- Shortness of breath
- Itching
- Hives
- Palpitations
- Respiratory Depression
- Cardiac Arrest
- Coma
- Seizures
- Death
Interestingly, it is impossible to tell what effect mixing this substance with Weed will have on an individual due to their own unique genetic make up and tolerance. It is never advisable to mix Choline Magnesium Trisalicylate and Weed due to the chances of mild, moderate and severe side effects. If you are having an adverse reaction from mixing Choline Magnesium Trisalicylate and Weed it’s imperative that you head to your local emergency room. Even mixing a small amount of Choline Magnesium Trisalicylate and Weed is not recommended.
Taking Choline Magnesium Trisalicylate and Weed together
People who take Choline Magnesium Trisalicylate and Weed together will experience the effects of both substances. Technically, the specific effects and reactions that occur due to frequent use of Choline Magnesium Trisalicylate and weed depend on whether you consume more weed in relation to Choline Magnesium Trisalicylate or more Choline Magnesium Trisalicylate in relation to weed.
The use of significantly more weed and Choline Magnesium Trisalicylate will lead to sedation and lethargy, as well as the synergistic effects resulting from a mixture of the two medications.
People who take both weed and Choline Magnesium Trisalicylate may experience effects such as:
- reduced motor reflexes from Choline Magnesium Trisalicylate and Weed
- dizziness from Weed and Choline Magnesium Trisalicylate
- nausea and vomiting due to Choline Magnesium Trisalicylate and Weed
Some people may also experience more euphoria, depression, irritability or all three. A combination of weed and Choline Magnesium Trisalicylate leads to significantly more lethargy which can easily tip over into coma, respiratory depression seizures and death.
Mixing weed and Choline Magnesium Trisalicylate
The primary effect of weed is influenced by an increase in the concentration of the inhibitory neurotransmitter GABA, which is found in the spinal cord and brain stem, and by a reduction in its effect on neuronal transmitters. When weed is combined with Choline Magnesium Trisalicylate this primary effect is exaggerated, increasing the strain on the body with unpredictable results.
Weed and Choline Magnesium Trisalicylate affects dopamine levels in the brain, causing the body both mental and physical distress. Larger amounts of Choline Magnesium Trisalicylate and weed have a greater adverse effect yet leading medical recommendation is that smaller does of Choline Magnesium Trisalicylate can be just as harmful and there is no way of knowing exactly how Choline Magnesium Trisalicylate and weed is going to affect an individual before they take it.
Taking Choline Magnesium Trisalicylate and weed together
People who take Choline Magnesium Trisalicylate and weed together will experience the effects of both substances. The use of significantly more Choline Magnesium Trisalicylate with weed will lead to sedation and lethargy, as well as the synergistic effects resulting from a mixture of the two medications.
People who take both weed and Choline Magnesium Trisalicylate may experience effects such as:
- reduced motor reflexes from Choline Magnesium Trisalicylate and weed
- dizziness from weed and Choline Magnesium Trisalicylate
- nausea and vomiting of the Choline Magnesium Trisalicylate
Some people may also experience more euphoria, depression, irritability or all three. A combination of weed and Choline Magnesium Trisalicylate leads to significantly more lethargy which can easily tip over into coma, respiratory depression seizures and death.
Weed Vs Choline Magnesium Trisalicylate
Taking Choline Magnesium Trisalicylate in sufficient quantities increases the risk of a heart failure. Additionally, people under the influence of Choline Magnesium Trisalicylate and weed may have difficulty forming new memories. With weed vs Choline Magnesium Trisalicylate in an individual’s system they become confused and do not understand their environment. Due to the synergistic properties of Choline Magnesium Trisalicylate when mixed with weed it can lead to confusion, anxiety, depression and other mental disorders. Chronic use of Choline Magnesium Trisalicylate and weed can lead to permanent changes in the brain22.G. Lafaye, L. Karila, L. Blecha and A. Benyamina, Cannabis, cannabinoids, and health – PMC, PubMed Central (PMC).; Retrieved September 27, 2022, from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5741114/.
Choline Magnesium Trisalicylate Vs Weed
Studies investigating the effects of drugs such as Choline Magnesium Trisalicylate and weed have shown that the potential for parasomnia (performing tasks in sleep) is dramatically increased when Choline Magnesium Trisalicylate and weed are combined. Severe and dangerous side effects can occur when medications are mixed in the system, and sleep disorders are a common side effect of taking weed and Choline Magnesium Trisalicylate together.
When a small to medium amount of weed is combined with Choline Magnesium Trisalicylate, sleep disorders such as sleep apnea can occur. According to the latest data from the US Centers for Disease Control and Prevention (CDC) most ER visits and hospitalizations caused by too much weed were associated with other substances such as Choline Magnesium Trisalicylate.
How long after taking Choline Magnesium Trisalicylate can I smoke weed or take edibles?
To avoid any residual toxicity it is advisable to wait until the Choline Magnesium Trisalicylate has totally cleared your system before taking weed, even in small quantities.
Overdose on Choline Magnesium Trisalicylate and weed
In the case of Overdose on Choline Magnesium Trisalicylate or if you are worried after mixing Choline Magnesium Trisalicylate and weed, call a first responder or proceed to the nearest Emergency Room immediately.
If you are worried about someone who has taken too much Choline Magnesium Trisalicylate or mixed weed with Choline Magnesium Trisalicylate then call a first responder or take them to get immediate medical help. The best place for you or someone you care about in the case of a medical emergency is under medical supervision. Be sure to tell the medical team that there is a mix of Choline Magnesium Trisalicylate and weed in their system.
Excessive Weed intake and result in scromiting, chs, and anxiety disorder. It is advisable to quit vaping weed if you are feeling these symptoms.
Mixing Choline Magnesium Trisalicylate and weed and antidepressants
Weed users feeling depressed and anxious may be prescribed antidepressant medication. There are some antidepressant users who also use Choline Magnesium Trisalicylate and weed. These individuals may not realize that there are side effects and consequences to consuming both Choline Magnesium Trisalicylate, marijuana and a range of antidepressants.
Studies on weed, Choline Magnesium Trisalicylate and antidepressants is almost nil. The reason for so little information on the side effects of the two is mostly down to marijuana being illegal in most places – although a number of states in the United States have legalized the drug.
Self-medicating with Weed and Choline Magnesium Trisalicylate
A lot of people suffer from depression caused by weed and Choline Magnesium Trisalicylate. How many? According to Anxiety and Depression Association of America (ADAA), in any given year, it is estimated that nearly 16 million adults experience depression. Unfortunately, that number is likely to be wrong due to under reporting. Many people do not report suffering from depression because they do not want to be looked at as suffering from a mental illness. The stigmas around mental health continue and people do not want to be labeled as depressed.
Potential side effects from mixing Choline Magnesium Trisalicylate and weed
Quitting weed to take Choline Magnesium Trisalicylate
Medical professionals say an individual prescribed or taking Choline Magnesium Trisalicylate should not stop using weed cold turkey. Withdrawal symptoms can be significant. Heavy pot users should especially avoid going cold turkey. The side effects of withdrawal from weed include anxiety, irritability, loss of sleep, change of appetite, and depression by quitting weed cold turkey and starting to take Choline Magnesium Trisalicylate.
A person beginning to use Choline Magnesium Trisalicylate should cut back on weed slowly. While reducing the amount of weed use, combine it with mindfulness techniques and/or yoga. Experts stress that non-medication can greatly improve a person’s mood.
Weed and Choline Magnesium Trisalicylate can affect a person in various ways. Different types of marijuana produce different side effects. Side effects of weed and Choline Magnesium Trisalicylate may include:
- loss of motor skills
- poor or lack of coordination
- lowered blood pressure
- short-term memory loss
- increased heart rate
- increased blood pressure
- anxiety
- paranoia
- increased energy
- increased motivation
Mixing Choline Magnesium Trisalicylate and weed can also produce hallucinations in users. This makes marijuana a hallucinogenic for some users. Weed creates different side effects in different people, making it a very potent drug. Now, mixing Choline Magnesium Trisalicylate or other mental health drugs with weed can cause even more unwanted side effects.
Mixing drugs and weed conclusion
Long-term weed use can make depression and anxiety worse. In addition, using marijuana can prevent Choline Magnesium Trisalicylate from working to their full potential33.J. D. Brown and A. G. Winterstein, Potential Adverse Drug Events and Drug–Drug Interactions with Medical and Consumer Cannabidiol (CBD) Use – PMC, PubMed Central (PMC).; Retrieved September 27, 2022, from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6678684/. Weed consumption should be reduced gradually to get the most out of prescription medication. Marijuana is a drug and it is harmful to individual’s long-term health. Weed has many side effects and the consequences are different to each person who uses it, especially when mixed with Choline Magnesium Trisalicylate.
If you take Choline Magnesium Trisalicylate, and also drink Alcohol or MDMA, you can research the effects of Choline Magnesium Trisalicylate and Alcohol , Choline Magnesium Trisalicylate and Cocaine as well as Choline Magnesium Trisalicylate and MDMA here.
To find the effects of other drugs and weed refer to our Weed and Other Drugs Index A to L or our Weed and Other Drugs Index M-Z.
Or you could find what you are looking for in our Alcohol and Interactions with Other Drugs index A to L or Alcohol and Interactions with Other Drugs index M to Z , Cocaine and Interactions with Other Drugs index A to L or Cocaine and Interactions with Other Drugs index M to Z or our MDMA and Interactions with Other Drugs Index A to L or MDMA and Interactions with Other Drugs Index M to Z.
Choline Magnesium Trisalicylate and Weed
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